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Pulmonary pathological changes and underlying mechanisms Due to systemic and pulmonary immunoinflammatory and immunothrombosis during ARDS, as well as subsequent disruption of the alveolar-capillary barrier, various cellular pathological changes, including various types of cell death, cellular senescence, and cellular dysfunction, have been demonstrated to occur in damaged pulmonary tissue

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The initial ischemic phase results in a scarcity of oxygen and nutrients, triggering cellular injury, oxidative stress, and inflammation (Snchez-Hernndez et al., 2020)

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During explosive activities like sprinting or weightlifting, phosphocreatine rapidly donates phosphate groups to regenerate ATP, fueling muscular contractions and increasing power output (Buford et al., Journal of the International Society of Sports Nutrition )

have more recently demonstrated Fas activation-induced eryptosis via ceramide formation in human erythrocytes exposed to CSE, as reported in the relevant paragraph [130]