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doi: 10.1097/00054725-200411000-00015
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DIE lesions also exhibit increased SK1, S1P 3 and S1P 5 mRNA levels and the expression of a specific S1P transporter, Spns2, responsible for the extracellular release of S1P, is altered only in DIE, further highlighting the dysregulation of S1P signalling in the different forms of endometriosis [15] (Fig
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[3] The same work profiled shifts in the cecal microbiome associated with treatment
Mode time-to-diagnosis for both adverse outcomes was 1-day post-admission
