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Description
This study observed a reduction in chronic pain and an improvement in sleep quality (see Castro-Marrero, J
The 4-HNE-induced ERK1/2 activation was mediated by repressing mitogen-activated protein kinase phosphatase-1 (MKP-1), a negative regulator of ERK1/2, through a proteasome-dependent degradation mechanism
When P r was calculated for the different substituents within the structures of interest, fragments with a tendency to form highly populated binding modes were identified, and hence, valuable substituent-interaction relationships could be determined
doi: 10.1152/ajpcell.00169.2023
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