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Formation of 4-HNE GCLC adduct increased the activity of monomeric GCLC but inhibited formation of GCL holoenzyme and lowered GCL holoenzyme activity [132]

Caspase-11 transcription and caspase-3 enzyme activity were both increased, suggesting that caspase may play a role in FaO cell death via valproic acid-mediated apoptosis (Phillips et al., 2003)

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