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The high-affinity NMDA-Rs on the beta cell are already saturated at physiological glutamate concentrations in the islet, and are mainly activated through depolarization of beta cells [121]
Flavahan, W
However, the efficiency of the ATP supply or utilization for GSH production seems to be very low because only 0.5 % of the total ATP regenerated in the glycolytic pathway is used for GSH production under anaerobic conditions [13]
This reduced the production of pro-atherogenic IL-1 110
Y.ZhangV
