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[79] Changes in CTNNB1 expression, which is the gene that encodes -catenin, can be measured in breast, colorectal, melanoma, prostate, lung, and other cancers

As a result, the dose to be administered to gain appreciable uPA/uPAR inhibition in vivo might be higher than can be tolerated in humans, which reduces clinical translation [171, 173]

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16 These molecules induce oxidative damage through unpaired valence-shell electrons that are highly unstable and reactive and have the ability to disrupt macromolecules, including proteins, lipids, carbohydrates, and nucleic acids

doi: 10.1016/j.nut.2012.11.012

Pharmacokinetics and pharmacokinetic-pharmacodynamic correlations of therapeutic peptides