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disruption of these protective antioxidants can also lead to oxidative stress This is a preview of subscription content, access via your institution Access options Subscribe to this journal Receive 12 print issues and online access $189.00 per year only $15.75 per issue Buy this article Purchase on SpringerLink Instant access to the full article PDF

Keywords: Antineoplastic agents, adverse effect, CIPN, clinical outcomes, animal study, mechanism, prevention and treatment 1

In the context of mast cell activation syndrome (MCAS) and dysautonomia, liposomal glutathione acts as a multi-targeted therapeutic tool

N-acetylcysteine increases manganese superoxide dismutase activity in septic rat diaphragms

Therefore, E2 can modulate both of these neuroendocrine systems by regulating NPY gene expression

Interleukin-6 reduces -cell oxidative stress by linking autophagy with the antioxidant response
