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This model demonstrates how interactions between APOA1 molecules can again be facilitated by a hinge-like H5 hairpin which is positioned at the apex of the trimeric assembly

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One possibility is that these patients may undergo delayed relapse, as has been described for a minority of patients years after achieving SVR.[6][20][21] Alternatively, these results suggest that some patients clear HCV RNA late, even after discontinuation of therapy (in those patients with an SVR who were HCV RNA-positive by TMA at the end of therapy)