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Liver fibrosis is a key marker of MASLD/MASH progression, initiated by elevated hepatocyte expression of pro-fibrotic TFs such as Sox9 and secreted factors such as Spp1 (osteopontin) and Tgfb1 (TGF-1)

Metabolic inhibition induces opening of unapposed connexin 43 gap junction hemichannels and reduces gap junctional communication in cortical astrocytes in culture

In the same animal model, the administration of the CB 1 receptor agonist ACEA decreased astroglial response in the vicinity of -amyloid plaques and decreased the expression of the pro-inflammatory cytokine interferon- in astrocytes ( Cannabis sativa L

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Hagen TM, Jones DP

Memantine for cognitive impairment in multiple sclerosis: a randomized placebocontrolled trial