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Collectively, these findings support a bidirectional feedback loop: Inflammatory cues (such as IL-1 and oxidative stress) suppress DNMT expression and activity, eroding genomic methylation, while low folate or MTHFR-limited flux and the resulting homocysteine/SAM:SAH imbalance promote oxidative and NF-B signaling

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Haqq AM, Chung WK, Dollfus H, Haws RM, Martos-Moreno G, Poitou C, et al

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doi: 10.1161/01.cir.103.22.2717 216 ZhangHSCaoEHQinJF

We also tested the thermal stability of the studied compounds