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For example, reduced NO synthesis further exacerbates oxidative stress and inflammatory responses, leading to the deterioration of endothelial injury (Levine et al., 2012

101 , 113 , 117 , 118 The particular illustration to damage counteraction appears with the ischemic/reperfusion colitis, 113 and rapid bypassing loop through arcade vessels

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found that significantly fewer particles were deposited in the lungs of PF patients compared to that deposited in the normal airway under equivalent conditions, based on the calculations of the fluid dynamics of simulated particles in the airway of PF patients [62], as portrayed in Fig