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Using gene knockdown and overexpression approaches, they further showed that silencing HIF-1 or HIF-2 significantly reduced hypoxia-induced LDHA expression (231)
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The success of this delayed treatment approach (dosing 48 h after the ischemic event) is clinically significant, as it models a feasible therapeutic window for intervention to mitigate secondary neuronal death following ischemic injury [97]
IAP-KO, IIAEK)
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For one, Arafa et al
