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In conclusion, OA pathogenesis is propelled by a metabolically dysregulated milieu, where obesity, oxidative stress, gut dysbiosis, and broader metabolic syndrome converge to create a self-perpetuating cycle of inflammation and tissue damage (Figure 1)

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Persistent malignant lymphadenopathy after preoperative chemoradiation identifies patients with poor postoperative survival

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Modulation of Multiple Pathways Involved in the Maintenance of Neuronal Function During Aging by Fisetin. Genes & Nutrition 4: 297307

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Moreover, in fresh samples from patients who underwent neoadjuvant chemotherapy, the GSH/GSSG levels in the resistant group were significantly higher than those in the sensitive group, which is consistent with previous research [28]

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The small molecule compound Fer-1 inhibits the occurrence of ferroptosis, inhibits the overexpression of MT1G, and restores the migration and invasion functions of gastric cancer cells through iron autophagy induced by the GPX4/SQSTM1 axis (Fig

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Please discuss any possible side effects with your doctor

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