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doi: 10.1039/D4TB00782D, [DOI] [PubMed] [Google Scholar] 262.Ghanim R, Kaushik A, Park J, Abramson A

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Melatonin administration reversed this phenotype by adopting a compensatory mechanism aimed at increasing Bcl-2 protein expression, blocking Bax proapoptotic activity via the SIRT1/NF-kB axis with a consequent and significant inhibition of Cyt C release and the lack of apoptosome formation and caspase 3 activation 37 (Fig

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