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This work also provides a mechanistically grounded, data-rich resource on FSP1 regulatory mechanisms and introduces new opportunities for targeting ferroptosis in cancer therapy
Redox environment of the cell and glutathione balance
doi: 10.1093/oxfordjournals.jbchem.a134444
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Finally, the different mechanisms of action for 5-FU and oxaliplatin may explain why 5-FU, but not oxaliplatin, increases H2AX caused by the inactivation of GSTA4
