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Salt generates antiinflammatory Th17 cells but amplifies their pathogenicity in proinflammatory cytokine microenvironments

This action supports the maintenance and repair of connective tissues subjected to mechanical stress

To improve disease symptoms, pulmonary rehabilitation involving patient-dependent exercises and breathing techniques plays a vital role

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Elevated production of ROS and an imbalance in antioxidant defense and repair mechanisms result in the loss or apoptosis of dopaminergic neurons in the substantia nigra [89]

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