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Additionally, LA may increase the efficiency of insulin signaling by regulating the expression and phosphorylation of IRS, subsequently decreasing hepatic lipid synthesis and storage

Tau hyperphosphorylation may result from OS-mediated interaction with tau protein kinase and phosphatase, particularly GSK-3 and PP2A, the predominant enzymes regulating Tau metabolism

For example, the histidine residue at position 43 (His-43) in SLC30A1 is important for the ability of this protein to transport Zn and is consistent with the corresponding His conserved in all other SLC30A members 88