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The antibodies used in this study are listed in Supplemental Table 1

Imai et al

Tanya HILDA

341,345 , we can hypothesize that aging-related NAD + decline might be the cause of mitochondria dysfunction

Knocking out the preproghrelin gene in mice does not seem to affect their size, growth rate, food intake, body composition, and reproduction, indicating that proghrelin products (acyl- or desacyl-ghrelin, obestatin) are not dominantly and critically involved in mouse viability, appetite regulation, and fertility (347), although subtle reductions in the amplitude of secretory GH peaks can be detected in these knockout mice during their youth: these differences recede with aging (348)