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METH stimulates the release of excess glutamate from neurons while inhibiting its uptake by astrocytes, leading to the overactivation of NMDARs ( tThe excitotoxicity induced by METH also triggers neuroinflammation and amplifies its neurotoxic effects

However, these observations have not been confirmed in large-scale human trials

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found that HSA formulations were stable for up to 36weeks at concentrations of 50% and higher of trehalose (Tower et al

The biggest risks include counterfeit products, contamination, incorrect dosing, poor storage, unknown ingredients, and lack of sterility