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For example, the overexpression of ANT2 can activate glycolysis and induce mitophagy, increasing ATP production and accelerating healing in aged skin wounds [133]

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[DOI] [PMC free article] [PubMed] [Google Scholar] 263.He L., Li J.-L., Zhang J.-J., Su P., Zheng S.-L

During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes