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The reasons for these discrepancies are unclear although a follow-up study from Erickson et al attributed the discrepant results to a lack of functional consequence in the ARE mutant used by Galloway and McLellan (Erickson et al., 2002)
References: Dunn MA et al (1997): Pretreatment for Nerve Agent Exposure (Chapter 6)
Line 2 is 20610, the joint injection with no modifier
Inflammatory processes can promote the generation of ROS, while oxidative stress can also activate proinflammatory signaling pathways [116]
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