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When the shield is down, oxidative stress rises, and cells become vulnerable
Similarly, in models of 6-OHDA-induced or spontaneous cell death, UA pretreatment prevented the activation of ROS-related death pathways and increased cell viability [169, 170]
To further investigate the efficiency of A80.2HCl action on the MYC, KLHL42 and pRB1 axes identified in our study, we treated bladder cancer, prostate cancer and breast cancer cell lines with increasing doses of A80.2HCl
doi: 10.1371/journal.pone.0229566 172 KvietkauskasMZitkuteVLeberBStrupasKStieglerPSchemmerP
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Presentation through MHC-II of native cellular autoantigens or viral EBNA-1 (Fig
