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Description
GLUD1 mutations reduce the sensitivity of the enzyme to its inhibitor GTP, which fuels hyperinsulinaemia/hyperammonaemia (HI/HA) syndrome, a condition characterized by elevated blood NH4 + levels and low glucose levels (MacMullen et al., 2001)
For the kaolin-intake study in Sprague-Dawley rats, 8-week-old double-housed male rats were used
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doi: 10.1152/ajpendo.00054.2012
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