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Given that glutaryllipoyl adducts form under conditions of glutarate excess on another TCA cycle ketoacid dehydrogenase, the PDHc 9 , we hypothesized that the major role of ABHD11 may be to reverse lipoyl glutarylation, maintaining TCA cycle integrity (Fig

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(2016) Treatment strategies in Alzheimers disease: a review with focus on selenium supplementation, Biometals , 29 , 827-839, Stoffaneller, R., and Morse, N

Guerfel, M., Baccouri, O., Boujnah, D., Chaibi, W

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10.1111/j.1365-2362.2011.02609.x 151