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Selenoprotein P and apolipoprotein E receptor-2 interact at the blood-brain barrier and also within the brain to maintain an essential selenium pool that protects against neurodegeneration

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This functional deficit contrasts sharply with the normal physiological role of microglia, which is to rapidly respond to neuronal injury signals and clear cellular debris

This dataset could be expanded to include other aliphatic and aromatic amino acids in the future

Abstract 2391: microglia-stimulated glioblastoma cell invasion is dependent on the EGFR ligand amphiregulin

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