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This hypothesis was supported by a marked increase in NF-B activation in embryonic fibroblast from HSF-1-null mutant mice due to lack of HSPs expression [62]

The expression of AKR1B15, a novel enzyme that shares 92% amino acid sequence homology with AKR1B10, is generally low in most tissues and limited to the adipose tissue, testis, and placenta [16]

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Soft tissue repair models

doi:10.1177/11206721221106149

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