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During necrosis, excessive Ca 2+ inflow causes mitochondrial calcium overload, and ROS destroys lipid, protein, and DNA structures, which jointly lead to disordered ionic equilibrium and mitochondrial dysfunction

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Exposure to OTA can down-regulate the formation of GCLC, the rate-limiting enzyme in GSH synthesis, which will result in a reduction in the intracellular level of GSH [73]

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