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2, in c-Met signaling, overexpression of the c-Met receptor and HGF results in sustained PI3K/Akt/mTOR and MAPK signaling activation through Gab1 and mutation of MET kinase domain, which are associated with therapy resistance [87]

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AOD-9604 appears to reproduce a portion of growth hormones lipolytic signaling in fat tissue increasing lipolytic sensitivity, raising fat oxidation, and restoring suppressed 3-AR expression toward normal in obese animals without engaging the growth-hormone receptor in the classical high-affinity way and without raising IGF-1