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For instance, GluN2A-NMDAR predominant entorhinal-CA1 synapses show depressed NMDAR excitatory postsynaptic potentials (EPSCs), whereas GluN2B-NMDAR predominant CA3-CA1 inputs have potentiated NMDAR EPSCs from D1-like receptor activation [150]

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The most likely cause of the fatigue is l ack of production of creatine due to functional vitamin B12 deficiency (see below)