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The function of glutamine antagonists in cancer cells is to induce intracellular hypoxia, acidosis and nutrient deprivation in cancer cells, whereas in effector T cells, glutamine antagonists promote intracellular antioxidant metabolism and induce transformation of T cells into long-lived, high-activity and high-memory subtypes

Cells under a higher partial pressure of oxygen are permanently exposed to attack by ROS which is balanced by the concerted action of a sophisticated antioxidant defense system (Bandyopadhyay et al

The activation of Nrf2 increased the enzymatic activities of SOD and GPx by inducing the expression of HO-1, and simultaneously inhibited the generation of lipid peroxidation marker MDA, thereby exerting an antioxidant effect (Zhang et al., 2022)

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Progressive decline in avoidance learning paralleled by inflammatory neurodegeneration in transgenic mice expressing interleukin 6 in the brain

targeting lysosomes in PDT) 70,71