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212 FBXO31 enhances cisplatin-induced ferroptosis in cholangiocarcinoma cells and stem cell-like cells, and the mechanism may be related to increased GPX4 ubiquitination and proteasome degradation, which could be reversed by GPX4 overexpression

doi: 10.1016/j.jns.2008.08.021

In positron emission tomography (PET)/CT, the high rate of glycolysis in NSCLC is reflected by the high uptake of 18F-fluorodeoxyglucose at the corresponding tumor sites (12)

& Tateyama, Y

This is largely nonsense, and potentially dangerous nonsense at that

The assembly of cargo/CPP interaction via non-covalent interaction strategy appears to be advantageous because it simplifies chemical conjugation protocols