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Common variants of the PYROXD2 gene are associated with small increases of TMA in urine [21], and it is logical to assume that variants exist that might be more disruptive and have commensurately greater effects on TMA accumulation
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Profile of the bovine acute-phase response following an intravenous bolus-dose lipopolysaccharide challenge
In conclusion, oxidative stress can be a possible explanation of hepatic damage induced by chronic therapy with chlorpheniramine maleate and cetirizine hydrochloride
Very recently, a study shows itaconate is required for the activation of the anti-inflammatory transcription factor Nrf2 (also known as NFE2L2) by lipopolysaccharide in mouse and human macrophages via dicarboxylation of KEAP1 (Mills et al., 2018)
