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Meanwhile, stress-response molecules, such as NRF2, glutathione peroxidase 2 (GPX2), aldoketo reductases (AKRs), ubiquitin C-terminal hydrolase L1 (UCHL1) and glutathione S-transferase mu 3 (GSTM3), upregulate PD-L1 to promote immune evasion in melanoma [69, 70]