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Therefore, IRE1s RIDD activity in mediating cellular sensitivity to ferroptosis is unique to IRE1 and functions as an important regulatory component of ferroptosis

No, it's a-How do you do it
A recent excellent review has highlighted the ongoing challenges in using GSH/GSSG titration as a measure of thiol redox balance and the best pre-analytical and analytical methods for the quantification of these molecules in biological samples [37]

Our findings were echoed in another clinical study, in which Lactobacillus enriched in GD patients was found to be associated with thyroid autoimmune antibodies (67)

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Some work, some did not work