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In animal models, studies show that the lack of PINK1 resulted in abnormal mitochondrial morphology, loss of SNpc DA neurons, reduction in complex I activity, and enhanced vulnerability to oxidative stress (Yang et al., 2006

The PCA score plots showed no obvious separations among the groups at varying time points (Figure 6A)

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10.1126/science.1176009 [DOI] [PMC free article] [PubMed] [Google Scholar] Jacquet M., Guittaut M., Fraichard A., Despouy G