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10.3390/bioengineering8070090 [DOI] [PMC free article] [PubMed] [Google Scholar] Figueiredo Neto M., Liu S., Salameh J

It is suggested that the role of selenium in aging may be due to selenoprotein involvement in redox homeostasis and anti-inflammatory activities (Legrain et al., 2014) and low levels of selenium can result in shortened telomere length (a hallmark of senescence), which can be rescued by selenium administration (Schoenmakers et al., 2010

For example, curcumin appears to cause DNA damage at both the mitochondrial and nuclear levels, suggesting that it may have a carcinogenic effect at doses near those that show positive results

As previously described, the classical proinflammatory categories (i.e., TLR, MAPK, LPS, and IFN signaling) were enriched in the downregulated clusters (12, 35)

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Studies suggest that glutathione is safe when taken correctly and at recommended dosages