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Since then, studies have shown that the overexpression of PKC drives tumor development via synergistic activation of several cell-survival and mitotic pathways, including nuclear factor-B (NF-B), signal transducer and activator of transcription 3 (Stat3), phosphatidylinositol 3-kinase/protein kinase B (PI3K/Akt), and extracellular signal-regulated kinase (ERK) [151,152]

Macrophage polarization in intestinal inflammation and gut homeostasis

Scaffidi P (2016) Histone H1 alterations in cancer

Ellagic acid prevents cisplatin-induced oxidative stress in liver and heart tissue of rats

Activated TBK1 phosphorylates the C-terminal tail (CTT) of STING at several residues including Ser366, which is part of a highly conserved motif (pLXIS) (143, 144)

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