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The morphological changes and biochemical processes that differentiate ferroptosis from other forms of programmed cell death are indicative of the unique characteristics of this process (Lee et al
Bhawan J, Grimes P, Pandya AG, Keady M, Byers HR, Guevara IL, et al
In rats, GCLC has a Km for glutamate that is about 10-fold higher than that of the GCL holoenzyme, which is higher than the cellular glutamate concentration in most tissues GCL is specific for the glutamyl moiety and is regulated physiologically by: (a) non-allosteric feedback competitive inhibition by glutathione (GSH) (Ki = 2.3 mM) which involves binding of GSH to the glutamate and another site on the enzyme [8, 9] and (b) availability of its precursor, cysteine [1]
They also significantly influence drug metabolism, affecting the pharmacokinetics of various therapeutic agents
Antioxid Redox Signal 24(7):376391 Dauer W, Przedborski S (2003) Parkinsons disease: mechanisms and models
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