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It is conceivable that there is a high possibility of acquired resistance after treatment with KRAS (G12C) inhibitors 228
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The aforementioned activation may consequently result in the release of substance P, neurokinin A, or calcitonin gene-related peptide (CGRP) [16]
doi: 10.7759/cureus.35244
10.1523/jneurosci.23-13-05928.2003 J
The consequent depletion of glutathione [3] impedes the efficient quenching of free radicals, leading to redox imbalance and oxidative stress [4]
