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An oncogenic role for alternative NF-kappaB signaling in DLBCL revealed upon deregulated BCL6 expression
The underlying molecular mechanism involves the activation of CRH-1/CREB by CMK-1/CaMKI in AFD neurons, which in turn upregulates the FMRFamide neuropeptide (FLP-6) that acts on AIY interneurons [49] (Figure 1)
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Moreover, the current study is only a retrospective study that has a less causal relationship than a cohort study
Decrease in vitamin C concentration in human lenses during cataract progression
