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Pharmacol Rep [Internet]

This suppression is dependent on the RNase activity of IRE1, as expression reconstitution with wild-type IRE1 but not the K907A mutant in IRE1-null cells inhibited GCLC expression (Fig
Mutalik, C

The mean deviation is also compared with the mean variability of the measurements of the gold standard

The result is broader brain coverage of appetite regulation circuits than either agent alone

Journal of Inorganic Biochemistry
