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These findings suggest that enhanced oxidative stress, coupled with impaired antioxidant defense mechanisms such as NQO1 suppression, plays a central role in mercury-induced pulmonary damage
The primary antibodies include Glutamate cysteine ligase catalytic and modifier subunits (GCLC, GCLM), Glutathione Synthetase (GSS), Peroxisome proliferator-activated receptor-gamma coactivator (PGC)1alpha (PGC1), mitochondrial complexes I-V, mitochondrial adenosine triphosphate (ATP) synthase F1 subunit alpha (ATP5A), Phosphate and tensin homolog (PTEN)-induced kinase 1 (PINK1)
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Genetic predisposition to the disease is observed widely
