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No clear phenotype has been associated with CAV2 deficiency in humans, while Cav2 knockout mice demonstrate severe pulmonary dysfunction along with little or no change in caveolin-1 expression and caveolae formation [46]

The best quality evidence is that which demonstrates significant positive outcomes, on human subjects, in multiple, double-blinded controlled studies

Myeloperoxidase and serum amyloid A contribute to impaired in vivo reverse cholesterol transport during the acute phase response but not group IIA secretory phospholipase A(2)

The pathophysiology, diagnosis, and treatment of dry eye disease