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Monocyte ADAM17 promotes diapedesis during transendothelial migration: identification of steps and substrates targeted by metalloproteinases J Immunol 190 8: 4236-44

Sympathomimetic drugs (e.g., phenylephrine, pseudoephedrine) and parasympatholytic drugs (e.g., atropine, ipratropium) dilate the pupil and thus pose a risk of angle closure through this mechanism

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After release, TML is hydroxylated into 3-hydroxyl-6-N-trimethyl-lysine (HTML) by trimethyl dioxygenase (TMLD), which is then broken down into 4-N-trimethyl-butylaldehyde (TMABA) and glycine by HTML aldolase (HTMLA) (1)

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