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10.1093/jn/125.4.823 [DOI] [PubMed] [Google Scholar] Ozoani H., Ezejiofor A
Our findings revealed that Gstp gene knocked caused a significant increase in mouse serum HMGB1 level, suggesting that GSTP might function as a negative regulator in controlling HMGB1 release in sepsis
London, UK: 2019
A simple foil wrap eliminates this concern entirely
The demonstration of phosphorylation of GCL using cultured hepatocytes suggests phosphorylation-dephosphorylation may be an important physiologic regulator of GCL
Front Mol Neurosci 2017
