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Both influenza-induced neutrophil dysfunction and neutrophil-independent mechanisms contribute to increased susceptibility to a secondary Streptococcus pneumoniae infection

By inhibiting this complex, Rh-2025u further suppresses the migration of CAFs (Di Donato et al., 2021a)
In fact, it has been shown that UDCA pre-treatment protects cultured rat hepatocytes against oxidative stress induced by ROS, significantly increasing the total amount of GSH and thiol-containing proteins, via the up-regulation of GCL mRNA expression and metallothionein [187]
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