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Recent studies in the rat model of WD, found that methanobactin, a peptide produced by proteobacterium Methylosinus trichosporium , can be successfully used to remove excess Cu from mitochondria, decrease liver histopathology, and prevent liver failure ( Since dysregulation of nuclear receptors, especially LXR and FXR, contribute significantly to WD phenotype (see above), targeting these receptors, could be an attractive alternative option for patients who do not respond or poorly tolerate Cu chelation

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For many, these signs might lead them to wonder about their vitamin B12 levels

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All methods were reported in accordance with ARRIVE guidelines