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Catabolism of extracellular glutathione by -glutamyltransferases supports tumour growth and survival, and pharmacological targeting of these enzymes slows tumour growth.

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Nuclear factor kappa B (NF-κB) plays a central role in the inflammatory process, and activation of Keap1-Nrf2 system counteracts with NF-κB by upregulating various genes such as superoxide dismutase (SOD), catalase, and heme oxygenase-1 (HO-1)