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The gutbrain axis and the microbiome: mechanisms and clinical implications

The central theme is the self-perpetuating cycle of airway dysfunction, inflammation, infection, and structural damage [4]

As MOTS-c activates AMPK and NRF2, it also indirectly suppresses the mTOR (mechanistic target of rapamycin) pathway, the master growth and anabolism regulator that, when chronically overactivated, accelerates the accumulation of senescent cells

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This dissociation suggests that some prosocial effects of oxytocin may be mediated by downstream metabolites acting through noncanonical mechanisms beyond OXTR activation