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Several studies have shown that the HGF/c-MET pathway participates in the recruitment and increase of MDSCs through multiple ways, and then promotes tumor immune evasion

Studies demonstrate that chronic microglial activation correlates with sustained neuroinflammation, potentially accelerating neurodegeneration through oxidative stress induction, immune dysregulation and apoptotic pathway activation (15,16)

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Furthermore, PSAT1 regulates changes in the level of glutamine-derived -KG, which controls mESC pluripotency and differentiation 106

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